Table S4: from No evidence that a transmissible cancer has shifted from emergence to endemism in Tasmanian devils

Copy number variants.Genome coordinates, relative to mSarHar1.11 [7], of 155 copy number variants (CNVs) identified in one or more DFT1 tumours presented by Patton et al. [14]. Each CNV is defined as a “gain” or “loss”. CNVs associated with the unstable marker 5 chromosome (“IsMarker5”) [6,7], or occurring on a region of chromosome 5 prone to copy number instability in DFT1 (“IsRecurrent”) are labelled. Genotypes for 48 tumours presented by Patton et al. [14] are shown (1, presence; 0, absence). Three tumours (SAMN14418891 [T-1560], SAMN14418906 [T-223262], SAMN14418921 [T-695064]) were excluded due to insufficient DFT1 sequencing coverage. Visualisation of autosomal absolute copy number profile for each tumour is presented in supplementary data S2.

拷贝数变异（Copy number variants, CNVs）：本数据集包含Patton等人[14]在1个或多个DFT1肿瘤中鉴定得到的155个拷贝数变异（CNVs）的基因组坐标，该坐标以参考基因组mSarHar1.11 [7]为参照。每个CNV被定义为"扩增（gain）"或"缺失（loss）"。与不稳定5号染色体标记（"IsMarker5"）[6,7]相关，或位于DFT1中易发生拷贝数不稳定的5号染色体区域的CNVs（"IsRecurrent"）均已完成标注。Patton等人[14]报道的48例肿瘤的基因型信息已展示（1代表存在，0代表缺失）。3例肿瘤（SAMN14418891 [T-1560]、SAMN14418906 [T-223262]、SAMN14418921 [T-695064]）因DFT1测序覆盖度不足被排除。每例肿瘤的常染色体绝对拷贝数谱可视化结果详见补充数据S2。