Flt3-ITD drives cooperating mutation-specific changes in cell identity and variable interferon dependence [RNA-seq dt2]

RNA-seq on NHD13/Flt3-ITD AML cells on Ifnar1 wildtype and kncokout backgrounds HPCs were from mice of the indicated genotypes

本研究针对处于干扰素α/β受体1（Ifnar1）野生型与敲除两种背景下的NHD13/Flt3-ITD急性髓系白血病（AML）细胞（即造血祖细胞，Hematopoietic Progenitor Cells，HPCs）开展RNA测序（RNA-seq），实验所用细胞均取自携带指定基因型的小鼠。