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BMI1 inhibition improves lesion burden in cerebral cavernous malformations

ChEBI2026-01-01 更新2026-07-12 收录

神经血管疾病治疗

基因靶向治疗

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-13726 数据链接链接失效反馈

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资源简介：

BMI1 inhibition improves lesion burden in cerebral cavernous malformations

应用场景：

创建时间：

2026-01-01

糖尿病肾病治疗

基因靶向治疗

The ER-stress response and in particular the maladaptive and cell-death-promoting transcription factor CCAAT/Enhancer-Binding Protein Homologous Protein (CHOP) have been linked with diabetic kidney di

NIAID Data Ecosystem50

RNA enzyme-directed gene therapy.

RNA基因治疗

基因靶向治疗

RNA enzyme-directed gene therapy.

PubMed Central1993-12-01 更新30

Design of a novel triple helix-forming oligodeoxyribonucleotide directed to the major promoter of the c-myc gene

基因靶向治疗

核苷类似物药物设计

Altered expression of c-myc is implicated in pathogenesis and progression of many human cancers. Triple helix-forming oligonucleotides (TFOs) directed to a polypurine/polypyrimidine sequence in a crit

PubMed Central2002-06-15 更新20

sequencing of C57BL/6J male mice after injecting with APAP and treating with si-Cyp2e1 LNPs

药物性肝损伤模型

基因靶向治疗

C57BL/6J male mice were injected with 300 mg/kg APAP to establish DILI model and were given si-Cyp2e1 LNPs via the tail vein for treatment.

NIAID Data Ecosystem40

Targeting the 3D genome by anthracyclines for chemotherapeutic effects

基因靶向治疗

蒽环类药物作用机制

The chromatins are folded into three-dimensional (3D) structures inside cells, which coordinates the regulation of gene transcription by the non-coding regulatory elements. Aberrant chromatin 3D foldi

NIAID Data Ecosystem30

BMI1 inhibition improves lesion burden in cerebral cavernous malformations

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