Transcriptional and Phenotypic Reprogramming of Tumors by Host Stroma Deficient in TNFα Signaling

ChEBI2010-07-20 更新2026-07-21 收录

肿瘤微环境

TNF信号通路

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-23002 数据链接链接失效反馈

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资源简介：

Transcriptional and Phenotypic Reprogramming of Tumors by Host Stroma Deficient in TNFα Signaling

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2010-07-20

Table_1_Comprehensive Genomic Characterization of Tumor Microenvironment and Relevant Signature in Clear Cell Renal Cell Carcinoma.xlsx

肿瘤微环境

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PurposeTo systematically investigate the characterization of tumor microenvironment (TME) in clear cell renal cell carcinoma (ccRCC), we performed a comprehensive analysis incorporating genomic altera

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Natural killer (NK) cells are circulating lymphocytes that possess both innate and adaptive features, the latter including antigen-specific clonal expansion and long-lived memory responses. Unlike oth

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Basal-to-inflammatory transition enhances basal cell carcinoma (BCC) therapy resistance via crosstalk with a pro-inflammatory stromal niche

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肿瘤微环境

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem00

Table_1_Risk Signature of Cancer-Associated Fibroblast–Secreted Cytokines Associates With Clinical Outcomes of Breast Cancer.xlsx

癌症预后预测

肿瘤微环境

Cancer-associated fibroblasts (CAFs) are key components in tumor microenvironment (TME). The secreted products of CAFs play important roles in regulating tumor cells and further impacting clinical pro

NIAID Data Ecosystem30

DataSheet1_Poly I:C-priming of adipose-derived mesenchymal stromal cells promotes a pro-tumorigenic phenotype in an immunocompetent mouse model of prostate cancer.zip

肿瘤微环境

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Introduction: Mesenchymal stromal cells (MSC) are envisioned as a potential cellular vehicle for targeted cancer therapies due to their tumor tropism and immune permissiveness. An obstacle in their us

NIAID Data Ecosystem00

Transcriptional and Phenotypic Reprogramming of Tumors by Host Stroma Deficient in TNFα Signaling

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