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Eccentric exercise activates novel transcriptional regulation of hypertrophic signaling pathways

ChEBI2009-12-16 更新2026-07-21 收录

肌肉肥大分子机制

运动生理转录调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-19062 数据链接链接失效反馈

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Eccentric exercise activates novel transcriptional regulation of hypertrophic signaling pathways

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创建时间：

2009-12-16

肌肉肥大分子机制

SIRT蛋白功能

SIRT5 is one of the seven members of the NAD+-dependent sirtuin family of protein deacylase that is mainly present in mitochondria and regulates metabolism. In heart, SIRT5 is highly expressed and res

NIAID Data Ecosystem80

RNA expression profiling in cardiomyocytes. RNA expression profiling in cardiomyocytes

肌肉肥大分子机制

RNA表达谱分析

Using the highly sensitive RNA array, we assessed the effect of 11β-HSD1 and BVT.2733 on modulating RNAs expression in the neonatal rat ventricular cardiomyocytes. Overall design: In this study, sampl

NIAID Data Ecosystem50

Transcriptome analysis of satellite cells isolated from an animal model of skeletal muscle hypertrophy

肌肉肥大分子机制

细胞转录组学

Skeletal muscle is a post-mitotic tissue with unique plasticity. It has an extraordinary ability to adjust to changes in its physiological environment by altering its metabolic phenotype and its mass.

NIAID Data Ecosystem30

H4K20me1 and gene expression in adult myocyte hypertrophy

肌肉肥大分子机制

表观遗传调控

The lysine methyltransferase KMT5A is shown to mediate the effects of CDC14A in the adult cardiomyocyte by regulating H4K20 mono-methylation, such that reduced KMT5A expression promotes a phenotype re

NIAID Data Ecosystem60

Murine bone marrow-derived mast cells (BMMCs) transduced with pAPM lentiviruses overexpressing miR-221 vs control overexpressing miR-221m.

miR-221功能研究

肌肉肥大分子机制

Bone marrow-derived mast cells were derived from three different female C57Bl/6 mice of similar age. Cells were transduced with pAPM lentiviruses either overexpressing microRNA-221 or a non functional

NIAID Data Ecosystem50

Eccentric exercise activates novel transcriptional regulation of hypertrophic signaling pathways

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