Defining Gene Signatures for Protective Immunity to Tuberculosis in the Chinese Cynomolgus Macaque Model

ChEBI2012-11-15 更新2026-07-21 收录

牛结核病与副结核病免疫机制

非人灵长类动物

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-42273 数据链接链接失效反馈

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Defining Gene Signatures for Protective Immunity to Tuberculosis in the Chinese Cynomolgus Macaque Model

创建时间：

2012-11-15

Copy Number Variation of CCL3- like Genes Affects Rate of Progression to Simian-AIDS in Rhesus Macaques ( Macaca mulatta )

艾滋病易感性遗传学

非人灵长类动物

Variation in genes underlying host immunity can lead to marked differences in susceptibility to HIV infection among humans. Despite heavy reliance on non-human primates as models for HIV/AIDS, little

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Data_Sheet_2_Infection of Monocytes From Tuberculosis Patients With Two Virulent Clinical Isolates of Mycobacterium tuberculosis Induces Alterations in Myeloid Effector Functions.zip

牛结核病与副结核病免疫机制

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Monocytes play a critical role during infection with Mycobacterium tuberculosis (Mtb). They are recruited to the lung, where they participate in the control of infection during active tuberculosis (TB

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Table_2_Alternative splicing of pre-mRNA modulates the immune response in Holstein cattle naturally infected with Mycobacterium avium subsp. paratuberculosis.xlsx

牛结核病与副结核病免疫机制

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Little is known about the role of alternative splicing (AS) in regulating gene expression in Mycobacteria-infected individuals in distinct stages of infection. Pre-mRNA AS consists of the removal of i

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Conserved Immune Recognition Hierarchy of Mycobacterial PE/PPE Proteins during Infection in Natural Hosts

牛结核病与副结核病免疫机制

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The Mycobacterium tuberculosis genome contains two large gene families encoding proteins of unknown function, characterized by conserved N-terminal proline and glutamate (PE and PPE) motifs. The prese

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CD38+ Alveolar Macrophages mediate early control of M.tuberculosis proliferation in the lung [CITE-seq]. CD38+ Alveolar Macrophages mediate early control of M.tuberculosis proliferation in the lung [CITE-seq]

牛结核病与副结核病免疫机制

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Tuberculosis, caused by M.tuberculosis (Mtb), remains an enduring global health challenge, especially given the limited efficacy of current therapeutic interventions. Much of existing research has foc

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Defining Gene Signatures for Protective Immunity to Tuberculosis in the Chinese Cynomolgus Macaque Model

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