This study evaluated the neuroprotective effects of recombinant HGF (rHGF) and endogenously induced HGF via N-Acetyl-Heparin (NAH) in a rat model of cerebral ischemia-reperfusion.
Endogenous neuroprotective mechanisms by which the brain protects itself against noxious stimuli and recovers from ischemic damage are key targets of stroke research, ultimately facilitating functiona
we used an electrical stimulation device to stimulate the rats for 1 h and then generated a rat model of ischemia/reperfusion. Ischemia/reperfusion was performed for 3, 6, 12, 24, or 72 h (5 rats at e
