() PPAR? mRNA relative expression in pooled liver samples from analyzed groups normalized to GAPDH expression

Results are expressed as mean ± SEM percentage of mean values obtained from equal numbers of C57BL/6J and C3He/6J mice. *P &lt; 0.05. (). SCARB1 mRNA relative expression in liver samples from analyzed groups normalized to GAPDH expression. *P &lt; 0.05 (). ChIP with a PPARγ antibody of study groups was used to amplify a SCARB1 promoter fragment. ChIP figures percentages indicate increments or decrements regarding C57BL/6J mice fed HFHC diet. Data of C57BL/6J mice fed chow diet was established as 100%.<b>Copyright information:</b>Taken from "Rosiglitazone but not losartan prevents Nrf-2 dependent CD36 gene expression up-regulation in an atherosclerosis model"http://www.cardiab.com/content/7/1/3Cardiovascular Diabetology 2008;7():3-3.Published online 26 Feb 2008PMCID:PMC2266907.

实验结果以均值±标准误（standard error of the mean, SEM）的百分比形式呈现，该百分比基于等量C57BL/6J与C3He/6J小鼠的均值计算得到。*P < 0.05。（）

本研究分析各组肝脏样本中清道夫受体B类1型（SCARB1）mRNA的相对表达量，以甘油醛-3-磷酸脱氢酶（GAPDH）作为内参基因进行标准化。*P < 0.05（）。

采用过氧化物酶体增殖物激活受体γ（PPARγ）抗体对各组进行染色质免疫共沉淀（chromatin immunoprecipitation, ChIP），以扩增SCARB1启动子片段。染色质免疫共沉淀结果的百分比变化以喂食高脂高胆固醇（high fat high cholesterol, HFHC）饲料的C57BL/6J小鼠为参照。喂食普通饲料的C57BL/6J小鼠的数据被设定为100%。

**版权信息**：本文引自《罗格列酮而非氯沙坦可在动脉粥样硬化（atherosclerosis）模型中抑制核因子红细胞2相关因子2（Nrf-2）依赖的CD36基因表达上调》，原文发表于《心血管糖尿病学（Cardiovascular Diabetology）》2008;7():3-3，在线发表时间为2008年2月26日，PMCID：PMC2266907，链接：http://www.cardiab.com/content/7/1/3