BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer
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https://www.ncbi.nlm.nih.gov/sra/SRP388465
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To explore the mechanisms of EGFR/HER2 activation by BET inhibition, we analyzed the mRNA changes in response to BET inhibition. Overall design: Transcriptomic responses were profiled with BETi (JQ1) treatment in HCC1954, BT474 and SKOV3 cells using mRNA sequencing.
创建时间：
2022-10-25
