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Ablation of casein kinase 1 in keratinocytes induces p53-dependent hyperpigmentation of the skin
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创建时间：
2019-05-14
相关数据集
Single Turnover Autophosphorylation Cycle of the PKA RIIβ Holoenzyme
To provide tight spatiotemporal signaling control, the cyclic adenosine monophosphate (cAMP)-dependent protein kinase (PKA) holoenzyme typically nucleates a macromolecular complex or a “PKA signalosom
NIAID Data Ecosystem50
PKI overexpression in HEK293 cells. PKI overexpression in HEK293 cells
RNA sequencing (RNAseq) of unstimulated cells transiently expressing PKI1-24 or inPKI1-24 as a control. Overall design: HEK293 cells where transduced in triplicate with plasmids expressing for GFP fus
NIAID Data Ecosystem30
Tranexamic acid may promote melanocores clustering in keratinocytes through upregulation of Rab5b. Tranexamic acid may promote melanocores clustering in keratinocytes through upregulation of Rab5b
To explore the effect of TXA on melanocores in keratinocytes. Overall design: RNA profiles of melanocore-incorporated normal human epidermal keratinocytes (NHEK) treated by Tranexamic acid (TXA)，and m
NIAID Data Ecosystem10
The neuronal transcription factor NeuroD and its impact on the regulation of dendricity and pigmentation of melanocytes in postinflammatory hyperpigmentation of human skin. Homo sapiens
In order to test the gene expression profile in postinflammatory hyperpigmentation, micoarray studies were performed. Overall design: Totally 14 subjects ( 7 at each phase) were treated by suction bli
NIAID Data Ecosystem40
C. albicans strains used in this study.
Protein kinases are central components of almost all signaling pathways that control cellular activities. In the model organism Saccharomyces cerevisiae, the paralogous protein kinases Ypk1 and Ypk2,
Figshare2023-08-10 更新20
