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Adult cardiomyocyte specific loss of myostatin
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2014-05-16
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Effects on gene expression of ibrutinib treatment in human stem cells-derived atrial- and ventricular-like cardiomyocytes. Effects on gene expression of ibrutinib treatment in human stem cells-derived atrial- and ventricular-like cardiomyocytes
To gain further insight into the mechanisms underlying the different response of atrial- and ventricular-like cardiomyocytes to ibrutinib, we performed RNA-seq in ibrutinib- or vehicle-treated atrial
NIAID Data Ecosystem150
RNA-sequencing analysis of ZsYellow-negative cells and ZsYellow-positive cardiopharyngeal progenitors and cardiomyocytes purified from the same 14-16 somites stage Tg(nkx2.5:ZsYellow) zebrafish embryos
The identification of novel cardiomyocyte-intrinsic factors that support ventricular function will expand the number of candidate genes and therapeutic options for heart failure, a leading cause of de
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Functional enrichment of shared top 500 genes between RV-CMs isolated via Langendorff or TSAD.
Spreadsheet “kegg”: KEGG enrichment of shared top 500 genes between RV-CMs isolated via Langendorff or TSAD (related to Fig 3C). Spreadsheet “GO_BP”: GO enrichment of shared top 500 genes between RV-C
NIAID Data Ecosystem20
Transcriptional profiling identifies differential expression of long non-coding RNAs in Jo-1 associated and inclusion body myositis. Homo sapiens
Myositis is characterised by muscle inflammation and weakness. Although generally thought to be driven by a systemic autoimmune response, increasing evidence suggests that intrinsic changes in the mus
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Additional file 1 of DNA G-quadruplex profiling in skeletal muscle stem cells reveals functional and mechanistic insights
Additional file 1: Fig. S1. G4 profiling reveals dynamic remodeling of G4s during mMuSC lineage progression. Fig. S2. G4 profiling reveals dynamic remodeling of G4s during hMuSC activation. Fig. S3. G
Figshare2025-09-05 更新30
