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NFAT-dependent IL-2 and IL-23 produced by DC are key regulators of pathogenic (TH17-mediated lung inflammation)

ChEBI2015-12-21 更新2026-07-12 收录

TH17免疫应答

肺部炎症机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-58590 数据链接链接失效反馈

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NFAT-dependent IL-2 and IL-23 produced by DC are key regulators of pathogenic (TH17-mediated lung inflammation)

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创建时间：

2015-12-21

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RNA sequencing was performed to study the genomic effects of formoterol, a long acting β2-adrenoceptor agonist, alone and in combination with the pro-inflammatory cytokine, TNFα in human primary bronc

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Changes in inflammatory mediator expression of the lung.

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The table shows B coefficients (95% confidence intervals) of the linear regression. Animals which received solely Ringers' lactate were used as reference group. mRNA expression is expressed as a fold-

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Data_Sheet_1_CD44 Loss Disrupts Lung Lipid Surfactant Homeostasis and Exacerbates Oxidized Lipid-Induced Lung Inflammation.PDF

巨噬细胞脂质代谢

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Alveolar macrophages (AMs) are CD44 expressing cells that reside in the alveolar space where they maintain lung homeostasis by serving critical roles in immunosurveillance and lipid surfactant catabol

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DataSheet_3_IL-17RA receptor signaling contributes to lung inflammation and parasite burden during Toxocara canis infection in mice.pdf

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IL-17 is a cytokine produced by innate and acquired immunity cells that have an action against fungi and bacteria. However, its action in helminth infections is unclear, including in Toxocara canis in

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NFAT-dependent IL-2 and IL-23 produced by DC are key regulators of pathogenic (TH17-mediated lung inflammation)

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