A mitochondrial long-chain fatty acid oxidation defect in a mouse leads to dysregulation of plasma long-chain acylcarnitines, dysregulation of plasma amino acids, and an increased reliance on glucocorticoid signaling to maintain euglycemia during fasting. A mitochondrial long-chain fatty acid oxidation defect in a mouse leads to dysregulation of plasma long-chain acylcarnitines, dysregulation of plasma amino acids, and an increased reliance on glucocorticoid signaling to maintain euglycemia during fasting
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下载链接：
https://www.ncbi.nlm.nih.gov/bioproject/PRJNA774961
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资源简介：
This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
创建时间：
2021-10-27
