首页/数据集/miR-671-5p promotes epithelial-to-mesenchymal transition by downregulating FOXM1 expression in breast cancer

miR-671-5p promotes epithelial-to-mesenchymal transition by downregulating FOXM1 expression in breast cancer

ChEBI2014-10-16 更新2026-07-21 收录

乳腺癌分子机制

上皮-间质转化调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-62411 数据链接链接失效反馈

官方服务：

资源简介：

miR-671-5p promotes epithelial-to-mesenchymal transition by downregulating FOXM1 expression in breast cancer

应用场景：

创建时间：

2014-10-16

乳腺癌分子机制

RNA调控网络

BRCA-related miRNA sponge interaction network. The network includes 37076 miRNA sponge interactions. (XLSX 484Â kb)

NIAID Data Ecosystem70

The primer sequence.

乳腺癌分子机制

癌症药物靶点

BackgroundBreast cancer continues to be a predominant cause of female mortality globally, characterized by limited therapeutic options and substantial adverse effects. Artesunate (ART), a traditional

Figshare2025-08-04 更新40

Gene expression analyses of PR action in the mammary gland of ovariectomized mice

乳腺癌分子机制

孕酮信号通路

Beyond demonstrating a critical role for progesterone receptor signaling in normal mammary epithelial proliferation, the progesterone receptor knockout mouse disclosed the progesterone receptor along

NIAID Data Ecosystem30

Shc proteins in DMBA induced breast cancer

乳腺癌分子机制

基因编辑模型

The principal factors that control mammary tumor formation remain insufficiently defined. Shc adaptor proteins play an important role in signaling by three well-established biological markers for prog

NIAID Data Ecosystem70

Dietary ligands diindolylmethane and resveratrol result in diverse ERα signaling not seen after E2 and a subset of diindolylmethane mediated signaling needs concurrent AHR activation.. Dietary ligands diindolylmethane and resveratrol result in diverse ERα signaling not seen after E2 and a subset of diindolylmethane mediated signaling needs concurrent AHR activation.

乳腺癌分子机制

受体信号通路

Inhibitory crosstalk between estrogen receptor alpha (ER alpha ) and aryl hydrocarbon receptor (AHR) regulates 17-estradiol (E2)-dependent breast cancer cell signaling. ER alpha and AHR are transcript

NIAID Data Ecosystem40

miR-671-5p promotes epithelial-to-mesenchymal transition by downregulating FOXM1 expression in breast cancer

资源简介：

相关数据集