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IRAK1, IRAK4 and MYD88 knockout in BCBL1 cells.
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创建时间：
2020-03-03
相关数据集
TIRAP drives myelosuppression through an Ifnγ-Hmgb1 axis that disrupts the endothelial niche.
Purpose: The goals of this study are to compare the transcriptome profile of mouse hematopoietic stem/progenitor cells (HSPC) from the bone marrow overexpressing the innate immune adaptor protein TIRA
NIAID Data Ecosystem40
BRET50 values calculated from BRET saturation curves show MyD88 and TLR2 interactions are dependent on TIRAP concentration.
BRET50 values calculated from BRET saturation curves show MyD88 and TLR2 interactions are dependent on TIRAP concentration.
Figshare2018-08-23 更新30
MYO1F Positions cGAS on the Plasma Membrane to Ensure Full and Proper Functional Execution
Cyclic GMP–AMP synthase (cGAS) recognizes viral or endogenous DNA to activate the innate immune response to infection and autoimmune diseases. After binding double-stranded DNA, cGAS synthesizes 2′3′
Mendeley Data20
Gp130-dependent release of acute phase proteins is linked to the activation of innate immune signaling pathways. Mus musculus
Gp130 dependent gene expression was analyzed in a previously established hepatocyte-specific gp130-knockout mouse model. Whole transcriptome analysis for isolated hepatocytes was performed to measure
NIAID Data Ecosystem30
