The transcriptome sequencing on muscle tissue from control group (CTX) and denervated group(CTX-DeN)

NIAID Data Ecosystem2026-05-02 收录

肌肉疾病转录组学

骨骼肌异位骨化机制

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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1136075 数据链接链接失效反馈

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The transcriptome sequencing on muscle tissue from control group (CTX) and denervated group(CTX-DeN). The PI3K/Akt pathway is involved in denervation-aggravated muscle HO.

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2024-07-15

Table_2_Transcriptome Analysis of Immune Receptor Activation and Energy Metabolism Reduction as the Underlying Mechanisms in Interleukin-6-Induced Skeletal Muscle Atrophy.xlsx

骨骼肌萎缩机制

肌肉疾病转录组学

BackgroundInflammation may trigger skeletal muscle atrophy induced by cancer cachexia. As a pro-inflammatory factor, interleukin-6 may cause skeletal muscle atrophy, but the underlying molecular mecha

NIAID Data Ecosystem90

Next Generation Sequencing Facilitates Quantitative Analysis of primary myoblast cell treated with different concentration glycine Transcriptomes. Next Generation Sequencing Facilitates Quantitative Analysis of primary myoblast cell treated with different concentration glycine Transcriptomes

肌肉疾病转录组学

甘氨酸药物干预

Purpose: The goals of this study are to analyze primary myoblast cell from mdx mice skeletal muscle transcriptome profiling (RNA-seq) treated with different concentration glycine. Methods: primary myo

NIAID Data Ecosystem60

COPD is accompanied by coordinated transcriptional perturbation in the quadriceps affecting the mitochondria and ECM

肌肉疾病转录组学

线粒体-ECM基因调控

In this experiment we catalogue transcriptional changes accompanying COPD in the quadriceps. We measure global gene transcription in the quadriceps using Affymetrix HuGene 1.1 ST arrays in an unselect

NIAID Data Ecosystem40

Comparative profiling in 13 muscle disease groups. Homo sapiens

肌肉疾病转录组学

神经肌肉疾病比较分析

Summary: Genetic disorders of muscle cause muscular dystrophy, and are some of the most common inborn errors of metabolism. Muscle also rapidly remodels in response to training and innervation. Muscle

NIAID Data Ecosystem70

Chronic rare DUX4 expression in mouse drives transcriptional similarities to human FSHD in whole muscle and fibroadiopgenic progenitors. Chronic rare DUX4 expression in mouse drives transcriptional similarities to human FSHD in whole muscle and fibroadiopgenic progenitors

FSHD分子机制

肌肉疾病转录组学

RNA-seq data was obtained on various samples from the iDUX4pA mouse model for FSHD and from control WT littermates. Overall design: RNA-seq was performed on whole muscle, as well as sorted endothelial

NIAID Data Ecosystem60

The transcriptome sequencing on muscle tissue from control group (CTX) and denervated group(CTX-DeN)

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