Identify WT and p53 3KR target in basal progenitor cells of epidermis
收藏NIAID Data Ecosystem2026-05-02 收录
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE275726
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Unbalanced and growth promoting cell fate dynamics that follow p53 loss, and the inability of canonical p53 functions to restrict clonal expansion, suggest the existence of a non-canonical p53 transcriptional program that controls key cell fate regulators. We hypothesized that these cell fate genes are among the shared targets of p53WT and p533KR. p53 ChIP-seq performed in isolated epidermal basal progenitor cells of p53WT and p533KR animals, 25uM of Nutlin-3a were added to digestion medium to stabilize p53
创建时间：
2024-08-31
