Supplementary information files for Sedentary behaviour, physical activity and psychobiological stress reactivity: a systematic review

Supplementary files for article Sedentary behaviour, physical activity and psychobiological stress reactivity: a systematic review.
Background
Sedentary behaviour, physical activity, and psychobiological reactivity to acute psychological stress are independent risk factors for cardiovascular disease. Sedentary behaviour and physical activity influence autonomic, haemodynamic, and inflammatory pathways under resting conditions, and these pathways become activated under acute psychological stress. However, it is unclear whether sedentary behaviour and physical activity relate to psychobiological responses to stress. Thus, the aim of this study is to systematically review sedentary behaviour and physical activity in the context of psychobiological reactivity to acute psychological stress.
Methods
Sedentary behaviour, physical activity and psychobiological stress reactivity search terms were combined, and several databases were searched in duplicate. Eligibility criteria included: (1) a validated measure of sedentary behaviour/physical activity; (2) cardiovascular, inflammatory, neuroendocrine, or respiratory markers measured at rest and in response to laboratory-induced acute psychological stress.
Results
6084 articles were screened, with 11 included in a narrative synthesis. No studies measured postural components of sedentary behaviour, but 2/4 studies found that markers of sedentary behaviour (e.g., physical inactivity) were associated with elevated heart rate, dysregulated heart rate variability, or lowered cortisol responses to stress. Higher volumes of physical activity were linked to lower HR, cortisol, or immune responses to stress in 4/7 studies.
Conclusions
Extensive methodological variability precludes conclusions from being drawn. This review should be used to guide a more homogeneous and gold-standard literature, which accounts for postural components of sedentary behaviour using inclinometery, and the whole physical activity intensity spectrum using universal and reproducible approaches.

《久坐行为、身体活动及对急性心理压力的心理生物学应激反应：系统综述》的补充文件。

背景
久坐行为、身体活动以及对急性心理压力的心理生物学应激反应是心血管疾病独立的危险因素。在静息状态下，久坐行为和身体活动会影响自主神经、血流动力学和炎症通路，而在急性心理压力下，这些通路会被激活。然而，尚不清楚久坐行为和身体活动是否与对压力的心理生物学反应相关。因此，本研究旨在系统综述在心理生物学对急性心理压力反应的背景下，久坐行为和身体活动的研究。

方法
将久坐行为、身体活动和心理生物学应激反应的检索词相结合，并重复搜索了多个数据库。纳入标准包括：（1）对久坐行为/身体活动的有效测量；（2）在静息状态下及实验室诱导的急性心理压力下测量的心血管、炎症、神经内分泌或呼吸标志物。

结果
筛选了6084篇文章，其中11篇被纳入叙述性综述。没有研究测量久坐行为的姿势成分，但2/4项研究发现久坐行为的标志物（例如，身体不活动）与心率升高、心率变异性失调或应激下皮质醇反应降低相关。在4/7项研究中，较高的身体活动量与较低的心率、皮质醇或应激下的免疫反应相关。

结论
广泛的方法学变异性阻碍了结论的得出。本综述应被用于指导更统一和黄金标准的文献，该文献通过倾斜仪测量久坐行为的姿势成分，并通过通用和可重复的方法测量整个身体活动强度谱。