Additional file 1: of The lncRNA MIR2052HG regulates ERÎą levels and aromatase inhibitor resistance through LMTK3 by recruiting EGR1

Figshare2019-04-04 更新2026-04-29 收录

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https://figshare.com/articles/dataset/Additional_file_1_of_The_lncRNA_MIR2052HG_regulates_ER_levels_and_aromatase_inhibitor_resistance_through_LMTK3_by_recruiting_EGR1/7950698 数据链接链接失效反馈

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Table S1. Primers for LMTK3 CHIP assay. (XLSX 9 kb)

创建时间：

2019-04-04

Gene expression profile of patient-derived head and neck squamous carcinoma cells subjected to different treatments.. Gene expression profile of patient-derived head and neck squamous carcinoma cells subjected to different treatments.

头颈部鳞状细胞癌基因

化疗耐药机制

In order to investigate the mechanisms underlying response and resistance towards treatment in HNSCC, we used low passage in vitro HNSCC cultures from two patients, namely SJG26 and SJG17. Both cultur

NIAID Data Ecosystem70

Supplementary Table 5 from ERBB4-Mediated Signaling Is a Mediator of Resistance to PI3K and BTK Inhibitors in B-cell Lymphoid Neoplasms

化疗耐药机制

全外显子组变异分析

Whole exome sequencing data, including single nucleotide variants and copy number variants.

NIAID Data Ecosystem30

High-resolution Nanopore methylome-maps reveal random hyper-methylation at CpG-poor regions as driver of chemoresistance in leukemias.[RNA-Seq]

癌症表观遗传学

化疗耐药机制

Aberrant DNA-methylation at CpG dinucleotides is a hallmark of cancer and is associated with the emergence of resistance to anti-cancer treatment, though molecular mechanisms and biological signifi-

NIAID Data Ecosystem20

DataSheet2_Exogenous proline enhances susceptibility of NSCLC to cisplatin via metabolic reprogramming and PLK1-mediated cell cycle arrest.xlsx

化疗耐药机制

代谢重编程

The occurrence of cisplatin resistance is still the main factor limiting the therapeutic effect of non-small cell lung cancer (NSCLC). It is urgent to elucidate the resistance mechanism and develop no

NIAID Data Ecosystem40

Unveiling alterations of epigenetic modifications and chromatin architecture leading to lipid metabolic reprogramming during the evolutionary trastuzumab adaptation of HER2-positive breast cancer [Cut & Tag]. Unveiling alterations of epigenetic modifications and chromatin architecture leading to lipid metabolic reprogramming during the evolutionary trastuzumab adaptation of HER2-positive breast cancer [Cut & Tag]

化疗耐药机制

表观遗传调控

Secondary trastuzumab resistance represents an evolutionary adaptation of HER2-positive breast cancer during anti-HER2 treatment. Most current studies have uncovered the alternations of HER2 and assoc

NIAID Data Ecosystem20

Additional file 1: of The lncRNA MIR2052HG regulates ERÎą levels and aromatase inhibitor resistance through LMTK3 by recruiting EGR1

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