Chronic inflammation is epidemiologically linked to the pathogenesis of gastrointestinal diseases, including inflammatory bowel disease (IBD) and colorectal cancer (CRC). However, our understanding of

NIAID Data Ecosystem30

An enhancer-driven stem cell-like program mediated by SOX9 blocks intestinal differentiation in colorectal cancer [rnaseq_ls180]. An enhancer-driven stem cell-like program mediated by SOX9 blocks intestinal differentiation in colorectal cancer [rnaseq_ls180]

结直肠癌分子机制

RNA-seq转录组学

Genomic alterations that encourage stem cell activity and hinder proper maturation are central to the development of colorectal cancer (CRC). Key molecular mediators that promote these malignant prope

NIAID Data Ecosystem30

Metabolites more than 2-fold lower in hESC compared to hECC cells.

干细胞代谢

癌细胞代谢

Metabolites present in DMEM medium written in bold. 1Statistical significance was determined by a T-test with adjusted Bonferroni correction in MeV. P-values <0.05 marked as *, P-values <0.01 marked a

NIAID Data Ecosystem50

Next Generation Sequencing Facilitates Quantitative Analysis of Transcriptomes in negative control and miR-1290 knockdown HCT116 cells

结直肠癌分子机制

MicroRNA功能研究

Colorectal cancer (CRC) ranks third in incidence and second in mortality among cancers worldwide. Emerging researches showed that microRNAs (miRNAs) play an important role in CRC progression. Here, we

NIAID Data Ecosystem30

The ERRalpha metabolic nuclear receptor controls growth of colon cancer cells

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