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E2F7 represses a transcriptional network of oscillating cell cycle genes to control S-phase progression.

ChEBI2012-01-04 更新2026-07-21 收录

细胞周期调控

转录因子功能

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-32673 数据链接链接失效反馈

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E2F7 represses a transcriptional network of oscillating cell cycle genes to control S-phase progression.

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创建时间：

2012-01-04

细胞周期调控

环境胁迫信号转导

The bacterial cell cycle has been extensively studied under standard growth conditions. How it is modulated in response to environmental changes remains poorly understood. Here, we demonstrate that th

NIAID Data Ecosystem60

Arrest of G(1)-S Progression by the p53-Inducible Gene PC3 Is Rb Dependent and Relies on the Inhibition of Cyclin D1 Transcription

细胞周期调控

肿瘤抑制基因

The p53-inducible gene PC3 (TIS21, BTG2) is endowed with antiproliferative activity. Here we report that expression of PC3 in cycling cells induced accumulation of hypophosphorylated, growth-inhibitor

PubMed Central30

Additional file 9 of AP1 is a pioneer transcription factor that programmes cell fate through MADS-domain protein tetramerisation

转录因子功能

花发育调控

Additional file 9: Table S8. Complementation tables for AP1WT-GFP, AP1tet-GFP and AP1dm-GFP.

NIAID Data Ecosystem20

PU.1 promotes cell cycle exit in the murine myeloid lineage associated with down-regulation of E2F1. Mus musculus

髓系细胞分化

细胞周期调控

A doxycycline-inducible system was used to induce PU.1 expression in cultured myeloid cell lines. The parent cell line used was BN (Kamath et al., Leukemia 22:1214-1225, 2008). Overall design: RNA was

NIAID Data Ecosystem40

DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

癌症分子机制

细胞周期调控

The upregulated proline rich 11 (PRR11) plays a critical role in cancer progression. The relevant biological functions of PRR11 in pan-cancer development are not well understood. In the current study,

NIAID Data Ecosystem30

E2F7 represses a transcriptional network of oscillating cell cycle genes to control S-phase progression.

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