miR-181a modulates Acute Myeloid Leukemia susceptibility to natural killer cells

ChEBI2014-12-30 更新2026-07-21 收录

白血病免疫治疗

MicroRNA调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-2837 数据链接链接失效反馈

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miR-181a modulates Acute Myeloid Leukemia susceptibility to natural killer cells

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2014-12-30

Analysis of microRNA expression during Epstein-Barr virus encoded LMP1 and LMP2A transfected in TW03 cells. Homo sapiens

病毒致癌机制

MicroRNA调控

Epstein-Barr virus has been reported to regulate cellular microRNA expression in B cells. In the present study, we investigated the differential microRNAs modulated by Epstein-Barr virus in Naspharyng

NIAID Data Ecosystem90

Islet-specific microRNA changes driven by nutrient shift trigger postnatal beta-cell maturation - dispersed islet cell mRNAs after miR-17-5p transfection. Rattus norvegicus

胰岛β细胞

MicroRNA调控

We found that in rodents, postnatal beta-cell maturation is associated with changes in the expression of several islet microRNAs and discovered that these modifications are driven by changes in the nu

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LV-Induced Dendritic Cells for AML Immunotherapy

白血病免疫治疗

树突状细胞疫苗

Wilms’ tumor 1 antigen (WT1) is overexpressed in acute myeloid leukemia (AML). Unfortunately, clinical immunotherapeutic use of WT1 peptides against AML has been inconclusive. A tricistronic lentivira

NIAID Data Ecosystem40

Anti-miRs Competitively Inhibit microRNAs in Argonaute Complexes

MicroRNA调控

寡核苷酸药物作用机理

MicroRNAs (miRNAs), small RNA molecules that post-transcriptionally regulate mRNA expression, are crucial in diverse developmental and physiological programs and their misregulation can lead to diseas

NIAID Data Ecosystem90

Maternal recognition of pregnancy in the horse: are microRNAs the secret messengers? [RNA-seq]

马妊娠识别

MicroRNA调控

The signal for maternal recognition of pregnancy (MRP) has still not been identified in the horse. Endometrial biopsies, uterine fluid, embryonic tissues and yolk sac fluid were collected 13 days afte

NIAID Data Ecosystem50

miR-181a modulates Acute Myeloid Leukemia susceptibility to natural killer cells

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