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Mechanisms of Ikaros Tumor Suppression with HDAC1 through distinct histone markers in Pre-B ALL Leukemia

ChEBI2015-11-03 更新2026-07-12 收录

白血病表观遗传学

肿瘤抑制机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-44217 数据链接链接失效反馈

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Mechanisms of Ikaros Tumor Suppression with HDAC1 through distinct histone markers in Pre-B ALL Leukemia

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创建时间：

2015-11-03

白血病表观遗传学

染色质免疫沉淀测序

Genome occupancy profiling by high throughput sequencing ChIP-seq of Brd4 with or without Doxycycline treatment to induce Med12 or Med23 shRNAs in MLL-AF9 transformed acute myeloid leukemia cells (RN2

NIAID Data Ecosystem110

Additional file 7 of Loss of H3K27 methylation identifies poor outcomes in adult-onset acute leukemia

白血病表观遗传学

基因突变关联分析

Additional file 7: Table S3. Pairwise associations between driver mutations presented by Pearson's Correlation Coefficient (cor) and p-value (p).

NIAID Data Ecosystem70

PLTP is a p53 target gene with roles in cancer growth suppression and ferroptosis

肿瘤抑制机制

铁死亡调控

The tumor suppressor protein p53 suppresses cancer by regulating processes such as apoptosis, cell cycle arrest, senescence, and ferroptosis. Whereas numerous p53 target genes have been identified, o

NIAID Data Ecosystem50

Common Tumor Suppressive Signaling of Thyroid Hormone Receptor Beta in Breast and Thyroid Cancer Cells

肿瘤抑制机制

转录组学分析

The thyroid hormone receptor beta (TRÃ) is a tumor suppressor in multiple types of solid tumors, most prominently in breast and thyroid cancer. An increased understanding of the molecular mechanisms

NIAID Data Ecosystem90

Unraveling MLL1-fusion Leukemia: Epigenetic Revelations from an iPS Cell Point Mutation

白血病表观遗传学

干细胞基因编辑

Human iPS cells (WT ASE9203 cells) were obtained from Applied Stem Cell (ASC), and CRISPR-Cas9-mediated gene editing was completed by ASC using their proprietary CRISPR-Cas9 protocol to introduce the

NIAID Data Ecosystem60

Mechanisms of Ikaros Tumor Suppression with HDAC1 through distinct histone markers in Pre-B ALL Leukemia

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