Pharmacologic targeting of sirtuin and PPAR signaling improves longevity and mitochondrial physiology in respiratory chain complex I mutant Caenorhabditis elegans.
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Pharmacologic targeting of sirtuin and PPAR signaling improves longevity and mitochondrial physiology in respiratory chain complex I mutant Caenorhabditis elegans.
对去乙酰化酶(sirtuin)与过氧化物酶体增殖物激活受体(PPAR)信号通路实施药物靶向调控,可延长呼吸链复合体I突变型秀丽隐杆线虫(Caenorhabditis elegans)的寿命并改善其线粒体生理功能。



