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MOESM4 of Exhaustion of mitochondrial and autophagic reserve may contribute to the development of LRRK2 G2019S -Parkinson’s disease

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DataCite Commons2020-08-29 更新2024-07-27 收录
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https://springernature.figshare.com/articles/MOESM4_of_Exhaustion_of_mitochondrial_and_autophagic_reserve_may_contribute_to_the_development_of_LRRK2___G2019S__-Parkinson_s_disease/6465353
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Additional file 4: Table S2. Raw data of mitochondrial phenotype and autophagic print parameters. The following legend accompanies the table: Glu: glucose; Gal: galactose; NM-LRRK2 G2019S : Non-manifesting carriers of LRRK2 G2019S -mutation; PD- LRRK2 G2019S : patients with LRRK2 G2019S -mutation and clinically manifest PD; mtDNA: Mitochondrial DNA; mtRNA: Mitochondrial RNA; CI: Complex I; O2 consumption stimulated for CI: Oxygen consumption stimulated for Complex I substrates (pyruvate, malate and glutamate); CIV: Complex IV. Pa. P value when comparing NM-LRRK2 G2019S vs. controls; Pb. P value when comparing PD- LRRK2 G2019S vs. controls; Pc. P value when comparing NM-LRRK2 G2019S vs. PD-LRRK2 G2019S .
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创建时间:
2018-06-09
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