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Tuberculosis alters immune-metabolic pathways resulting in perturbed IL-1 responses

ChEBI2022-11-05 更新2026-07-12 收录

结核病免疫代谢

细胞因子反应失调

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-12379 数据链接链接失效反馈

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Tuberculosis alters immune-metabolic pathways resulting in perturbed IL-1 responses

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创建时间：

2022-11-05

结核病免疫代谢

硫化氢-结核病机制

The ubiquitous gasotransmitter hydrogen sulfide (H2S) has been recognised to play a crucial role in human health. However, a role for host H2S in pathogenesis has not yet been demonstrated. Using c

NIAID Data Ecosystem100

Additional file 3 of Untargeted metabolomics analysis reveals Mycobacterium tuberculosis strain H37Rv specifically induces tryptophan metabolism in human macrophages

宿主-病原体代谢互作

结核病免疫代谢

Additional file 3.

DataCite Commons2024-02-20 更新60

Transcriptomics of vitamin D treatment effects in Mycobacterium tuberculosis-infected THP-1 cells

结核病免疫代谢

宿主-病原体相互作用

Previous reports have shown low vitamin D serum levels and polymorphisms in the vitamin D receptor (VDR) to be associated with increased risk for TB. Given that 1α,25-dihydroxyvitamin D3 has a role in

NIAID Data Ecosystem30

M. tuberculosis Induces Potent Activation of IDO-1, but This Is Not Essential for the Immunological Control of Infection

结核病免疫代谢

宿主-病原体相互作用

Indoleamine 2,3-dioxygenesae-1 (IDO-1) catalyses the initial, rate-limiting step in tryptophan metabolism, thereby regulating tryptophan availability and the formation of downstream metabolites, inclu

NIAID Data Ecosystem80

Analyzing the impact of Mycobacterium tuberculosis infection on primary human macrophages

结核病免疫代谢

宿主-病原体相互作用

The success of Mycobacterium tuberculosis (Mtb) as pathogen is tightly linked to its ability to recalibrate host metabolic processes in infected host macrophages. Correspondingly, analysis of RNA-sequ

NIAID Data Ecosystem50

Tuberculosis alters immune-metabolic pathways resulting in perturbed IL-1 responses

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