Mutant p53 cooperates with ETS2 to promote etoposide resistance [ChIP-Seq]

ChEBI2012-05-02 更新2026-07-21 收录

化疗耐药机制

ChIP-seq分析

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-36749 数据链接链接失效反馈

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资源简介：

Mutant p53 cooperates with ETS2 to promote etoposide resistance [ChIP-Seq]

创建时间：

2012-05-02

H3K9acS10P (new abcam lot).Kc

组蛋白修饰定位

ChIP-seq分析

modENCODE_submission_3009 This submission comes from a modENCODE project of Gary Karpen. For full list of modENCODE projects, see http://www.genome.gov/26524648 Project Goal: We aim to determine the l

NIAID Data Ecosystem30

Mitomycin resistance in mammalian cells expressing the bacterial mitomycin C resistance protein MCRA

化疗耐药机制

丝裂霉素抗性蛋白功能

The mitomycin C-resistance gene, mcrA, of Streptomyces lavendulae produces MCRA, a protein that protects this microorganism from its own antibiotic, the antitumor drug mitomycin C. Expression of the b

PubMed Central1999-08-31 更新10

ChIP-seq analysis of Purpureocillium lavendulum H3K14 modification cultured on PDB or PDB+Ammonium Sulfate

真菌组蛋白修饰

ChIP-seq分析

ChIP-seq analysis of Purpureocillium lavendulum H3K14 modification cultured on PDB or PDB+Ammonium Sulfate Overall design: 1Ã108 fungal conidia were inoculated into PDB and PDB+(NH4)2SO4 liquid media

NIAID Data Ecosystem10

DataSheet9_Therapeutic Suppression of FAK-AKT Signaling Overcomes Resistance to SHP2 Inhibition in Colorectal Carcinoma.ZIP

化疗耐药机制

AKT信号通路

SHP2 mediates signaling from multiple receptor tyrosine kinases (RTKs) to extracellular signal-regulated kinase (ERK) and Ser and Thr kinase AKT, and its inhibitors offer an unprecedented opportunity

NIAID Data Ecosystem10

QuantSeq 3’ mRNA-sequencing.

化疗耐药机制

EGFR泛素化调控

A precise balance between ubiquitination and deubiquitination is crucial for cellular regulation. Ubiquitin-specific peptidase 54 (USP54), an active deubiquitinase (DUB), modulates the ubiquitination

Figshare2025-04-01 更新10

Mutant p53 cooperates with ETS2 to promote etoposide resistance [ChIP-Seq]

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