Rat LAD IR study

Alliance of Genome Resources2026-08-01 收录

心肌缺血再灌注损伤

大鼠心脏模型

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https://www.ncbi.nlm.nih.gov/gds/?term=GSE4105 数据链接链接失效反馈

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Keywords: rat heart ventricles, LAD - left anterior descending coronary artery, IR - ischemia-reperfusion

关键词：大鼠心室（rat heart ventricles）、左前降支冠状动脉（Left Anterior Descending Coronary Artery，LAD）、缺血-再灌注（Ischemia-Reperfusion，IR）

Biochanin A attenuates myocardial ischemia/reperfusion injury through the TLR4/NF-κB/NLRP3 signaling pathway

NLRP3信号通路

心肌缺血再灌注损伤

Abstract Purpose: Myocardial ischemia/reperfusion (Ml/R) injury is a leading cause of damage in cardiac tissues, with high rates of mortality and disability. Biochanin A (BCA) is a main constituent of

DataCite Commons2024-02-28 更新60

Metabolic Adaptation to a Disruption in Oxygen Supply during Myocardial Ischemia and Reperfusion Is Underpinned by Temporal and Quantitative Changes in the Cardiac Proteome

心肌缺血再灌注损伤

蛋白质组学分析

Despite decades of intensive research, there is still no effective treatment for ischemia/reperfusion (I/R) injury, an important corollary in the treatment of ischemic disease. I/R injury is initiated

NIAID Data Ecosystem30

DataSheet4_Verapamil Alleviates Myocardial Ischemia/Reperfusion Injury by Attenuating Oxidative Stress via Activation of SIRT1.ZIP

心肌缺血再灌注损伤

SIRT1信号通路药理

Myocardial ischemia/reperfusion (I/R) injury is a potential complication of ischemic heart disease after recanalization. One of the primary reasons for I/R injury is the excessive accumulation of reac

NIAID Data Ecosystem10

Ischemia and Reperfusion Induce Differential Expression of Calpastatin and Its Homologue High Molecular Weight Calmodulin-Binding Protein in Murine Cardiomyocytes

心肌缺血再灌注损伤

蛋白酶调控机制

In the heart, calpastatin (Calp) and its homologue high molecular weight calmodulin-binding protein (HMWCaMBP) regulate calpains (Calpn) by inhibition. A rise in intracellular myocardial Ca2+ during c

NIAID Data Ecosystem40

Factor Xa and factor IIa inhibitors differentially regulate inflammation in myocardial ischemia reperfusion injury [19-0267 RNA-seq]

心肌缺血再灌注损伤

凝血蛋白酶抑制剂药效

While thrombin is the key protease in regard to thrombus formation, other coagulation proteases, such as fXa or activated protein C (aPC), independently modulate intracellular signaling via partially

NIAID Data Ecosystem20

Rat LAD IR study

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