Additional file 4 of Pathological mechanisms and candidate therapeutic approaches in the hearing loss of mice carrying human MIR96 mutations
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https://springernature.figshare.com/articles/dataset/Additional_file_4_of_Pathological_mechanisms_and_candidate_therapeutic_approaches_in_the_hearing_loss_of_mice_carrying_human_MIR96_mutations/27274121/1
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Additional file 4: Table S3. 127 genes were significantly differentially expressed in both Mir96 +13G>A and Mir96 +14C>A homozygotes. Eighty-four were upregulated, 40 were downregulated, and 3 were upregulated in Mir96 +14C>A homozygotes and downregulated in Mir96 +13G>A homozygotes. Two hundred and one DEGs were unique to Mir96 +13G>A homozygotes, and 566 DEGs were unique to Mir96 +14C>A homozygotes. Only genes with significant differential expression (FDR < 0.05) are shown.
提供机构：
Moreno Pelayo, Miguel Angel; Williams, Gareth; Fernandez, Sergio; Chen, Jing; Steel, Karen P.; Duddy, Graham; Lachgar-Ruiz, Maria; Morin, Matias; Di Domenico, Francesca; Lewis, Morag A.
创建时间：
2024-10-22
