The androgen receptor induces a distinct transcriptional program in castration resistant prostate cancer in man

ChEBI2013-01-04 更新2026-07-21 收录

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The androgen receptor induces a distinct transcriptional program in castration resistant prostate cancer in man

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2013-01-04

AR-V7 targets the oncogenic genes in castration resistant prostate cancer. [ChIP]. AR-V7 targets the oncogenic genes in castration resistant prostate cancer. [ChIP]

To elucidate the AR-V7 role, we performed ChIP-seq on H3K4me1, H3K4me3, H3K27ac, and AR antibody recognizing the N-terminus of AR and AR splice variants (AR-Vs) using LNCaP and LNCaP95 cells. We showe

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MOF forms a positive feedback regulation with ERα to suppress endometrial cancer progression

MOF is a histone acetyltransferase specific for H4K16 acetylation. It has been demonstrated that MOF is lower expressed in series of human cancers. However, the molecular mechanism underlying the deta

NIAID Data Ecosystem80

DHT-dependent AR activity in LNCaP cells. Homo sapiens

AR transcriptional activity is regulated by DHT Overall design: DNA microarrays we employed to determine changes in gene expression caused by DHT treatment

NIAID Data Ecosystem70

Supplementary Table S1-S4 from Targeting the MIF/CXCR7/AKT Signaling Pathway in Castration-Resistant Prostate Cancer

Supplementary Tables

aacr.figshare.com2023-06-15 更新80

Systematic characterization of epigenetic modifying enzymes identifies KDM3B as a key regulator in castration resistant prostate cancer. Systematic characterization of epigenetic modifying enzymes identifies KDM3B as a key regulator in castration resistant prostate cancer

Androgen deprivation therapy (ADT) is the standard care for prostate cancer patients who fail surgery and radiotherapy. While initially effective, the cancer almost always recurs into a more aggressiv

NIAID Data Ecosystem70