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The androgen receptor induces a distinct transcriptional program in castration resistant prostate cancer in man

ChEBI2013-01-04 更新2026-07-21 收录

去势抵抗性前列腺癌

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数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-28219 数据链接链接失效反馈

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The androgen receptor induces a distinct transcriptional program in castration resistant prostate cancer in man

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创建时间：

2013-01-04

去势抵抗性前列腺癌

癌症耐药机制研究

Purpose: Resistance to androgen deprivation therapies is a major driver of mortality in advanced prostate cancer. Therefore, there is a need to develop new pre-clinical models that allow the investiga

NIAID Data Ecosystem50

RNA-Seq of LNCaP human prostate cancer cells with knockdown of MAP3K7 and/or CHD1

膀胱癌分子亚型

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Purpose: The goal of this study is to identify transcriptome-wide changes that occur in a human prostate cell line with knockdown of MAP3K7(TAK1) and CHD1, in the presence and absence of androgens. We

NIAID Data Ecosystem50

GR and ER co-activation alters the expression of differentiation genes and associates with improved ER+ breast cancer outcome

乳腺癌分子机制

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Analysis of MCF-7 cells treated for 4h with Ethanol, Estradiol (E2), Dexamethasone (Dex), or Estradiol + Dexamethasone (E2 + Dex) In estrogen receptor (ER)-negative breast cancer (BC), high tumor glu

NIAID Data Ecosystem30

Genome-wide AR binding under androgen deprivation condition upon TRAF4 overexpression. Genome-wide AR binding under androgen deprivation condition upon TRAF4 overexpression

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全基因组结合位点分析

We report that TRAF4-mediated AR ubiquitination alters AR genomic binding profile under androgen deprivation condition. Overall design: Examination of AR genome-wide binding in control and TRAF4 overe

NIAID Data Ecosystem50

Androgen receptor RIP-seq of LNCaP cells treated with androgen hormone. Homo sapiens

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lincRNA转录调控

In this work we reported in a high-throughput way (RIP-seq) the RNAs associated with androgen receptor after treatment with androgen hormone. We used a large compilation of lincRNAs to describe the di

NIAID Data Ecosystem50

The androgen receptor induces a distinct transcriptional program in castration resistant prostate cancer in man

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