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Recurrent amplifications of the histone methyltransferase SETDB1 in melanoma

ChEBI2012-03-17 更新2026-07-21 收录

黑色素瘤基因组学

表观遗传驱动因素

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-36579 数据链接链接失效反馈

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Recurrent amplifications of the histone methyltransferase SETDB1 in melanoma

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创建时间：

2012-03-17

黑色素瘤基因组学

突变谱分析

Primary mucosal melanoma (MM), which arise from melanocytes located in mucosal membranes, frequently present late and have a poor prognosis; ~14% 5-year survival rate. MM makes up only ~1.4% of all me

NIAID Data Ecosystem80

Exome and transcriptome sequencing for the identification of immunogenic mutations in a human melanom model

肿瘤新抗原

黑色素瘤基因组学

Somatic point mutations in tumors can lead to the generation of tumor-specific neoantigens which can be recognized by the patientsâ own T cells. Such immunogenic mutations were searched for in the h

NIAID Data Ecosystem60

Table S10 from Discovery and Validation of an Ancillary Genomic Test of Malignancy for Primary Melanocytic Tumors

黑色素瘤基因组学

肿瘤辅助诊断

Clinical-pathological features and sequencing metrics for each sample in the borderline cohort.

Figshare2025-09-02 更新40

Genomics of acral lentiginous melanoma

黑色素瘤基因组学

癌症驱动突变识别

Melanoma is the most aggressive type of skin cancer, causing about 75% of dermatological cancer deaths. Acral lentiginous melanoma (ALM) is the most common subtype of melanoma in admixed Latin America

NIAID Data Ecosystem70

Functional mutations form at CTCF-cohesin binding sites in melanoma due to uneven nucleotide excision repair across the motif. Homo sapiens

黑色素瘤基因组学

CTCF结合位点

CTCF binding sites are frequently mutated in cancer, but how these mutations accumulate and whether they broadly perturb CTCF binding is not well understood. We report that skin cancers exhibit a high

NIAID Data Ecosystem40

Recurrent amplifications of the histone methyltransferase SETDB1 in melanoma

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