Homo sapiens Epigenomics. Homo sapiens

NIAID Data Ecosystem2026-05-02 收录

多发性骨髓瘤表观遗传学

人类表观基因组学

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1110794 数据链接链接失效反馈

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Multiple myeloma (MM) is a malignant neoplasm originating in the plasma cell system, with a higher incidence in elderly individuals.

创建时间：

2024-05-12

BP_August_2016_Bisulfite-Seq_Multiple_Myeloma_on_GRCh38 - samples

DNA甲基化分析

多发性骨髓瘤表观遗传学

Bisulfite-Seq data for 5 Multiple Myeloma sample(s). 63 run(s), 7 experiment(s), 10 analysis(s) on human genome GRCh38. Part of BLUEPRINT release August 2016. Analysis documentation available at http:

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MMSET alters EZH2 Binding in Myeloma Cells

多发性骨髓瘤表观遗传学

EZH2ChIP-seq分析

We investigated EZH2 binding in the presence and absence of MMSET protein. MMSET overexpression in t(4;14)+ myeloma leads to global loss of H3K27 methylation and redistribution of EZH2 binding through

NIAID Data Ecosystem80

HRP2-MINA complex reprograms t(4;14) multiple myeloma epigenome to dictate chemosensitivity to proteasome inhibitors [ChIP-Seq]

多发性骨髓瘤表观遗传学

蛋白酶抑制剂耐药性

Changes in these genes are probably a consequence of aging, and the real regulators governing BMSC senescence and osteogenesis are still unclear.In the current study, we report that nucleosome assembl

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Additional file 2 of The DNA methylation landscape of multiple myeloma shows extensive inter- and intrapatient heterogeneity that fuels transcriptomic variability

多发性骨髓瘤表观遗传学

DNA甲基化异质性

Additional file 2 Table S2. Description of sequencing metrics for eRRBS data.

Figshare2021-08-10 更新80

ChIP-Seq of Histone Deacetylases and Cell Cycle-Regulators in Multiple Myeloma cells. Homo sapiens

多发性骨髓瘤表观遗传学

ChIP-seq转录分析

Transcription of oncogenes is regulated by histone deacetylases and cyclins. Some of these proteins are targetable with small molecules. This series contains ChIP-Seq targeting transcriptional regulat

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Homo sapiens Epigenomics. Homo sapiens

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