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FACT maintains pluripotency factor expression through gene-distal regulation in embryonic stem cells

NIAID Data Ecosystem2026-05-01 收录

胚胎干细胞多能性调控

表观遗传学与基因表达

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE181624 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2023-07-19

胚胎干细胞多能性调控

表观遗传

Distinct cell types emerge from embryonic stem cells through a precise and coordinated execution of gene expression programs during lineage commitment. This is established by the action of lineage spe

NIAID Data Ecosystem40

DNA methylation from a Type I restriction modification system influences gene expression and virulence in Streptococcus pyogenes [RNA-seq]

毒力调控

表观遗传学与基因表达

We used Illumina NGS to measure mRNA levels and perform subsequent differential expression analysis between wild type and RM system deficient strains of Streptococcus pyogenes Examination of mRNA leve

NIAID Data Ecosystem30

CARM1 Mediates Modulation of Sox2

胚胎干细胞多能性调控

转录因子精氨酸甲基化

Sox2 is a key component of the transcription factor network that maintains the pluripotent state of embryonic stem cells (ESCs). Sox2 is regulated by multiple post-translational modifications, includi

NIAID Data Ecosystem30

Gene expression analysis of subpopulations of mouse embryonic stem cells sorted based on Id1 and Nanog expression

胚胎干细胞多能性调控

基因表达谱分析

Analysis of gene expression in sorted subpopulations of mouse embryonic stem cells. We set out to investigate whether expression of Id1 in Nanog-low cells affected the expression of pluripotency facto

NIAID Data Ecosystem30

Mitotic bookmarking redundancy by nuclear receptors mediates robust post-mitotic reactivation of the pluripotency network

胚胎干细胞多能性调控

有丝分裂转录因子功能

Mitotic bookmarking transcription factors (TFs) are thought to mediate rapid and accurate post-mitotic gene reactivation. However, the loss of individual bookmarking TFs often leads to the deregulatio

NIAID Data Ecosystem30

FACT maintains pluripotency factor expression through gene-distal regulation in embryonic stem cells

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