官方服务：
资源简介：
Expression profiling experiment on Pkd1-mutant mice and mice induced with nephrotoxin DCVC
应用场景：
创建时间：
2019-05-14
相关数据集
Gene expression in early phases of Autosomal Dominant Polycystic Kidney Disease
Little is known about the genes involved in the initial cyst formation and disease progression in autosomal dominant polycystic kidney disease (ADPKD). To uncover the genetic determinants and molecula
NIAID Data Ecosystem70
Supplementary Material for: UPREGULATED ANGIOTENSIN IA RECEPTORS IN THE HYPOTHALAMIC PVN SENSITISE NEUROENDOCRINE VASOPRESSIN RELEASE AND BLOOD PRESSURE IN A RODENT MODEL OF POLYCYSTIC KIDNEY DISEASE.
Introduction: Angiotensin (Ang) II signalling in the hypothalamic paraventricular nucleus (PVN) via angiotensin type-1a receptors (AT1R) regulates vasopressin release and sympathetic nerve activity -
Figshare2022-06-02 更新40
Microscopy images of renal macrophages in the tissue environment and in 3D cultures
Microscopy images (confocal, brightfield) shown in the publication. We present here the unedited raw images in a .tif or .jpg format. The images are supplementary information for the various figures s
bonndata2025-10-08 更新20
Dissection of metabolic reprogramming in polycystic kidney disease reveals coordinated rewiring of bioenergetic pathways
Autosomal Dominant Polycystic Kidney Disease (ADPKD) is a genetic disorder caused by loss-of-function mutations in PKD1 or PKD2. Increased glycolysis is a prominent feature of the disease, but how it
NIAID Data Ecosystem40
Fatty acid oxidation is impaired in an orthologous mouse model of Autosomal Dominant Polycystic Kidney Disease
Background The major gene mutated in autosomal dominant polycystic kidney disease was first identified over 20 years ago, yet its function remains poorly understood. We have used a systems-based appro
NIAID Data Ecosystem40
