Neuron-specific changes in gene expression caused by NPC1 deficiency

ChEBI2016-07-27 更新2026-07-12 收录

神经退行性分子机制

溶酶体贮积症基因表达

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-75775 数据链接链接失效反馈

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资源简介：

Neuron-specific changes in gene expression caused by NPC1 deficiency

创建时间：

2016-07-27

Aging-Associated Modulation of UFMylation Impairs Proteostasis in C. elegans

衰老相关蛋白

神经退行性分子机制

The attachment of Post-Translational Modifications (PTMs) to proteins plays key roles in the regulation of the activity and stability of various proteins. Here we utilized the nematode Caenorhabditis

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Global gene profiling of (-)-Epigallocatechin-3-gallate (EGCG)-induced neuronal death. Mus musculus

神经退行性分子机制

药物神经保护

Alzheimer’s disease (AD) is the most common form of adult-onset dementia with severe intellectual deterioration and is characterised by the accumulation of the amyloid-β (Aβ) peptides and the presence

NIAID Data Ecosystem30

Brain Extracellular Matrix implications in multiple neurological disorders revealed through a meta-analysis of transcriptional changes

神经退行性分子机制

元转录组分析

Authors: Hagit Sadis1*, Wote Amelo Rike1*, David Peles1, Yara Hussein1, Tchelet Stern1, Irit Sagi2, Shani Stern1* Affiliations: 1Sagol Department of Neurobiology, Faculty of Natural Sciences, Univer

Zenodo2025-11-11 更新10

Transcriptomic analysis of ZNF91 knockout in XDP iPSCs

神经退行性分子机制

CRISPRi基因编辑

X-linked Dystonia-Parkinsonism (XDP) is a severe neurodegenerative disorder resulting from the insertion of an intronic SINE-Alu-VNTR (SVA) retrotransposon in the TAF1 gene. Recent research has reveal

NIAID Data Ecosystem30

Additional file 3 of Cytokine enrichment in deep cerebellar nuclei is contributed by multiple glial populations and linked to reduced amyloid plaque pathology

神经退行性分子机制

单细胞RNA测序

Additional file 3: Supplementary Data for snRNAseq and RNAseq experiments.

Figshare2024-09-11 更新10