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HPV integration mode
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创建时间：
2022-10-04
相关数据集
Human HPV16 positive CaSki cells: Control vs. virus oncogene e6/e7 silenced
Human HPV16 positive CaSki cells: Control vs. virus oncogene e6/e7 silenced
ChEBI2016-07-01 更新40
Effect of HPV E5 on tumor on immune response and tumor development
To investigate the functions of HPV16 E5, we established empty vector or HPV16 E5-transduced CAL-27 and DC2.4 cell lines. We then performed gene expression profiling analysis using data obtained from
NIAID Data Ecosystem30
Identification of deregulated pathways, key regulators, and novel miRNA-mRNA interactions in HPV-mediated transformation. [CGH cell lines-Agilent]. Identification of deregulated pathways, key regulators, and novel miRNA-mRNA interactions in HPV-mediated transformation. [CGH cell lines-Agilent]
Next to a persistent infection with high-risk human papillomavirus (HPV), molecular changes are required for the development of cervical cancer. To identify which molecular alterations drive carcinoge
NIAID Data Ecosystem20
Long non-coding RNA FAM83H-AS1 is regulated by human papillomavirus 16 E6 independently of p53 in cervical cancer cells
Global long non-coding RNA (lncRNA) expression profile in primary human keratinocytes post-expression of high-risk HPV-16 E6 oncoprotein Purpose: The goal of this study is to utlize high-throughput RN
NIAID Data Ecosystem30
HPV integration generates cellular super enhancer and functions as ecDNA to regulate genome-wide transcription. HPV integration generates cellular super enhancer and functions as ecDNA to regulate genome-wide transcription
Human papillomavirus (HPV) integration is a critical step in cervical cancer development, while the oncogenic mechanism in genome-wide transcriptional level is still poorly understood. In this study,
NIAID Data Ecosystem30
