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Phenotype specific analyses of p53 reveal distinct regulatory mechanism for chronically activated p53

ChEBI2015-03-02 更新2026-07-21 收录

p53调控机制

肿瘤抑制基因

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-53491 数据链接链接失效反馈

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Phenotype specific analyses of p53 reveal distinct regulatory mechanism for chronically activated p53

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2015-03-02

母细胞瘤遗传学

肿瘤抑制基因

Deletion of 1p is associated with poor prognosis in neuroblastoma, however selected 1p-intact patients still experience poor outcomes. Since mutations of 1p genes may produce deleterious functional ef

NIAID Data Ecosystem120

Additional file 2: Table S2. of Concordance of copy number loss and down-regulation of tumor suppressor genes: a pan-cancer study

肿瘤抑制基因

癌细胞研究

The enriched pathways and interactors for the 207 tumor suppress genes (TSGs) with frequent copy number loss (CNL). (XLSX 11 kb)

DataCite Commons2024-12-12 更新60

MYBL2 Is a Sub-haploinsufficient Tumor Suppressor Gene in Myeloid Malignancy

肿瘤抑制基因

髓系恶性肿瘤

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem50

Molecular Evolution of PTEN Pseudogenes in Mammals

哺乳动物伪基因进化

肿瘤抑制基因

Phosphatase and tensin homolog (PTEN) is a tumor-suppressor gene. PTEN pseudogene (PTENp) acts as an endogenous RNA, which regulates its parental gene by competitively binding to the 3’ UTR of PTEN ge

NIAID Data Ecosystem80

Transcription profiling of transfected A549 lung cancer cell line with new tumor supressor gen ARLTS1 in that shows a very low expression level in comparison with normal lung

肿瘤抑制基因

癌细胞转录组

We have transfected a new tumor suppresor gene named ARLTS1 in A549 lung cancer cell line that shows a very low expression level in comparison with normal lung. This line was transfected using the pMV

NIAID Data Ecosystem50

Phenotype specific analyses of p53 reveal distinct regulatory mechanism for chronically activated p53

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