首页/数据集/Clinical pediatric acute lymphoblastic leukemia samples after in vitro exposure to L-asparaginase

Clinical pediatric acute lymphoblastic leukemia samples after in vitro exposure to L-asparaginase

ChEBI2006-01-20 更新2026-07-21 收录

急性淋巴细胞白血病

药物体外反应

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-4071 数据链接链接失效反馈

官方服务：

资源简介：

Clinical pediatric acute lymphoblastic leukemia samples after in vitro exposure to L-asparaginase

应用场景：

创建时间：

2006-01-20

相关数据集

B-ALL relapse analysis. Homo sapiens

急性淋巴细胞白血病

RNA表达分析

A comparison of RNA expression profiles from leukemic cells of 8 single B-ALL patients at diagnosis and relapse. For all patients, clonality of the blast cells at diagnosis and relapse was demonstrate

NIAID Data Ecosystem40

Association results of SNPs in CDKN2A/B and B-ALL.

急性淋巴细胞白血病

遗传关联分析

Association results of SNPs in CDKN2A/B and B-ALL.

Figshare2017-05-09 更新30

Myb overexpression synergizes with loss of Pten and is a dependency factor and therapeutic target in T-cell acute lymphoblastic leukemia

急性淋巴细胞白血病

MYC靶向治疗

T-lineage acute lymphoblastic leukemia (T-ALL) is an aggressive hematological malignancy that accounts for 10%â15% of pediatric and 25% of adult ALL cases. Although the prognosis of T-ALL has improv

NIAID Data Ecosystem20

A review of outcomes presented in published studies of pediatric acute lymphoblastic leukemia: Toward the development of acore outcome set.

急性淋巴细胞白血病

临床结局研究

Inlcuded is all the data from our study including the extraction sheet, the centrality measures, the matrices, and superstructure for all outcomes.

Figshare2016-07-13 更新30

Mechanistic study of Wee1 kinase inhibition with AZD1775 exposes drug targetable vulnerabilities in acute B-lymphoblastic leukemia. Mechanistic study of Wee1 kinase inhibition with AZD1775 exposes drug targetable vulnerabilities in acute B-lymphoblastic leukemia

Wee1激酶抑制机制

急性淋巴细胞白血病

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem30