A Staphylococcus pro-apoptotic peptide induces acute exacerbation of pulmonary fibrosis

NIAID Data Ecosystem2026-03-11 收录

脏器纤维化病理机制

细菌肽与宿主互作

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A Staphylococcus pro-apoptotic peptide induces acute exacerbation of pulmonary fibrosis

创建时间：

2020-04-22

Hippo signaling impairs alveolar epithelial regeneration in pulmonary fibrosis

脏器纤维化病理机制

上皮再生

Idiopathic pulmonary fibrosis (IPF) consists of fibrotic alveolar remodeling and progressive loss of pulmonary function. Genetic and experimental evidence indicates that chronic alveolar injury and fa

DataONE2023-05-23 更新30

Single cell RNA sequencing of cultured CCR10+ and CCR10- lung airway epithelial cells. Single cell RNA sequencing of cultured CCR10+ and CCR10- lung airway epithelial cells

单细胞转录组学

脏器纤维化病理机制

Mechanically dissociated IPF lung explant derived cellular suspensions were cultured in 50% senescent lung fibroblast conditioned medium + 50% fibroblast complete medium (DMEM + 15% FBS + antiboitics)

NIAID Data Ecosystem20

Animal model of acute-on-chronic alcoholic liver injury [DDC study]

酒精性肝炎动物模型

脏器纤维化病理机制

Background and aims: Liver is a major target organ for alcohol-induced disease and the spectrum of pathological states elicited by alcohol in liver comprises steatosis, alcoholic steatohepatitis, prog

NIAID Data Ecosystem10

Mapping spatially resolved transcriptomes in human and mouse pulmonary fibrosis

脏器纤维化病理机制

空间转录组学

Idiopathic pulmonary fibrosis (IPF) is a progressive lung disease with poor prognosis and limited treatment options. Efforts to identify effective treatments are thwarted by limited understanding of I

NIAID Data Ecosystem20

Regulation of macrophage foam cell formation during nitrogen mustard (NM)-induced pulmonary fibrosis by lung lipids

脏器纤维化病理机制

脂质代谢调控

Nitrogen mustard (NM) is a vesicant known to target the lung, causing acute injury which progresses to fibrosis. Evidence suggests that activated macrophages contribute to the pathologic response to N

DataONE2019-09-17 更新20

A Staphylococcus pro-apoptotic peptide induces acute exacerbation of pulmonary fibrosis

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