Browning of human adipocytes requires KLF11 and reprogramming of PPARγ super-enhancers

ChEBI2014-12-12 更新2026-07-21 收录

脂肪细胞褐变机制

分子代谢调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-59703 数据链接链接失效反馈

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资源简介：

Browning of human adipocytes requires KLF11 and reprogramming of PPARγ super-enhancers

创建时间：

2014-12-12

Table_2_Glucocorticoid-Responsive Transcription Factor Krüppel-Like Factor 9 Regulates fkbp5 and Metabolism.XLSX

糖皮质激素信号转导

分子代谢调控

Krüppel-like factor 9 (Klf9) is a feedforward regulator of glucocorticoid receptor (GR) signaling. Here we show that in zebrafish klf9 is expressed with GR-dependent oscillatory dynamics in synchrony

NIAID Data Ecosystem100

High-resolution structure of unliganded PPAR gamma in complex with the peptide PGC-1 alpha

受体结构生物学

分子代谢调控

High-resolution structure of unliganded PPAR gamma in complex with the peptide PGC-1 alpha Descriptor: Peroxisome proliferator-activated receptor gamma, Peroxisome proliferator-activated receptor gamm

Protein Data Bank Japan2024-02-07 更新30

Table_3_Effects of allicin on human Simpson-Golabi-Behmel syndrome cells in mediating browning phenotype.xlsx

脂肪细胞褐变机制

天然化合物药理

IntroductionObesity is a major health problem because it is associated with increased risk of cardiovascular disease, diabetes, hypertension, and some cancers. Strategies to prevent or reduce obesity

NIAID Data Ecosystem20

Additional file 1 of Transcriptional and metabolic effects of aspartate-glutamate carrier isoform 1 (AGC1) downregulation in mouse oligodendrocyte precursor cells (OPCs)

少突胶质前体细胞转录组学

分子代谢调控

Additional file 1: Full results of differential expression analysis.

Figshare2024-03-29 更新30

The long noncoding RNA lnc-ob1 facilitates bone formation by upregulating Osterix in osteoblasts

分子代谢调控

骨质疏松症靶点

Long noncoding RNAs (lncRNAs) have emerged as integral regulators of physiology and disease, but specific roles of lncRNAs in bone disease remain largely unknown. Here, we show that lnc-ob1 regulates

NIAID Data Ecosystem30

Browning of human adipocytes requires KLF11 and reprogramming of PPARγ super-enhancers

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