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BPTF is required for c-MYC-induced remodelling of target chromatin and transcriptional activation [ChIP-Seq]

ChEBI2016-01-19 更新2026-07-12 收录

癌症表观遗传学

染色质免疫沉淀测序

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-65544 数据链接链接失效反馈

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BPTF is required for c-MYC-induced remodelling of target chromatin and transcriptional activation [ChIP-Seq]

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创建时间：

2016-01-19

植物表观遗传学

染色质免疫沉淀测序

Analysis of H3.3 position and abundance in the presence and absence of a functionnal ATRX protein: we wanted to analyse how the absence of the Arabidopsis putative ATRX chaperone would impact the abun

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Mapping Context-Aware Phosphosite Regulation of Protein–Protein Interactions Using Deep Learning and Pan-Cancer Proteomics

磷酸化位点调控蛋白互作预测

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Phosphorylation dynamically orchestrates the protein–protein interaction (PPI) network that governs cellular signaling, and its dysregulation frequently drives malignant transformation and neurodegene

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CENPA as a reader of m6A-modified centromeric RNA to ensure centromere integrity in cancer cells [RIP-Seq]

癌症表观遗传学

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Maintaining centromere integrity is crucial for genome stability and proper chromosome segregation during mitosis. CENPA, a conserved histone H3 variant, is localized at the centromeres and plays an e

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Summary of OPCML methylation in cell lines and primary tumors.

癌症表观遗传学

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Summary of OPCML methylation in cell lines and primary tumors.

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Genome-wide analysis of SRC-1/NCOA1 and Erα/ESR1 binding in chromatin obtained from the ERα stably transfected U2OS osteosarcoma cell line (U2OS-ERα) with ChIP-SEQ.. Homo sapiens

受体信号通路

染色质免疫沉淀测序

To better understand the role of SRC-1 in estrogen signaling in bone, a genome-wide analysis of ERα and SRC-1 binding sites in the absense and presence of E2 was conducted. Overall design: Cells were

NIAID Data Ecosystem20

BPTF is required for c-MYC-induced remodelling of target chromatin and transcriptional activation [ChIP-Seq]

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