Cytomegalovirus-induced oncomodulation drives immune escape in glioblastoma. Cytomegalovirus-induced oncomodulation in glioblastoma

NIAID Data Ecosystem2026-05-02 收录

病毒诱导肿瘤调节

胶质瘤免疫逃逸

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Cytomegalovirus-induced oncomodulation drives immune escape in glioblastoma

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2025-01-29

Inhibition of ICAM1 diminishes stemness and enhances antitumor immunity in glioblastoma by regulating β-catenin/PD-L1 signaling pathway

胶质瘤免疫逃逸

免疫治疗靶点

Glioblastoma (GBM) stem cells (GSCs) are pivotal in tumor initiation, recurrence, and therapeutic resistance, underscoring their critical role in the complex pathology of GBM. Despite their recognized

NIAID Data Ecosystem110

Proteomic Comparison of 3D and 2D Glioma Models Reveals Increased HLA‑E Expression in 3D Models is Associated with Resistance to NK Cell-Mediated Cytotoxicity

胶质瘤免疫逃逸

NK细胞毒性抵抗

Three-dimensional cell culture techniques can better reflect the in vivo characteristics of tumor cells compared with traditional monolayer cultures. Compared with their 2D counterparts, 3D-cultured t

NIAID Data Ecosystem50

Glioblastoma cells acquire myeloid-affiliated transcriptional programs via epigenetic immunoediting to elicit immune evasion [WGS]

胶质瘤免疫逃逸

免疫表观遗传学

Glioblastoma multiforme (GBM) is an aggressive brain tumour. Current immunotherapy approaches have been unsuccessful, highlighting the need to determine underlying mechanisms of immune evasion. Here,

NIAID Data Ecosystem30

Glioblastoma cells acquire myeloid-affiliated transcriptional programs via epigenetic immunoediting to elicit immune evasion [BiSulfite-seq]. Glioblastoma cells acquire myeloid-affiliated transcriptional programs via epigenetic immunoediting to elicit immune evasion [BiSulfite-seq]

胶质瘤免疫逃逸

表观遗传免疫编辑

Glioblastoma multiforme (GBM) is an aggressive brain tumour. Current immunotherapy approaches have been unsuccessful, highlighting the need to determine underlying mechanisms of immune evasion. Here,

NIAID Data Ecosystem80

C1-inactivator is upregulated in glioblastoma

胶质瘤免疫逃逸

补体系统抑制

Background Glioblastoma is the most common and aggressive type of primary brain tumor in adults. A key problem is the capacity of glioma cells to inactivate the body’s immune response. The complement

NIAID Data Ecosystem40

Cytomegalovirus-induced oncomodulation drives immune escape in glioblastoma. Cytomegalovirus-induced oncomodulation in glioblastoma

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