RNA-Seq of mouse adrenal medulla and SDHB/NF1 and NF1 pheochromocytomas
收藏NIAID Data Ecosystem2026-03-13 收录
官方服务:
资源简介:
This study aimed to describe the transcriptomic profile of mouse SDHB-null pheochromocytomas by comparing to normal adrenal medulla and NF1 pheochromocytomas.
应用场景:
创建时间:
2021-12-21
相关数据集
Gene expression changes in HEI-193 (NF2-deficient Schwannoma) cells by Nf18001 or TEW7197 treatment.
Loss of NF2 (merlin) has been suggested as a genetic cause of neurofibromatosis type 2 and malignant peripheral nerve sheath tumor (MPNST). Previously, we demonstrated that NF2 sustained TGF- recepto
NIAID Data Ecosystem40
Table_5_SETDB1 Overexpression Sets an Intertumoral Transcriptomic Divergence in Non-small Cell Lung Carcinoma.XLSX
An increasing volume of evidence suggests that SETDB1 plays a role in the tumorigenesis of various cancers, classifying SETDB1 as an oncoprotein. However, owing to its numerous protein partners and th
NIAID Data Ecosystem10
Transcriptome profiles of malignant peritoneal mesotheliomas of rats induced by multiwalled carbon nanotubes (MWCNTs) and amosite asbestos
Malignant mesothelioma is an aggressive cancer that often originates in the pleural and peritoneal mesothelium. Exposure to asbestos is a frequent cause. But studies in rodents showed that certain mul
NIAID Data Ecosystem00
Transcriptomic profiling associated with temozolomide (TMZ) response of glioblastoma mouse model. Mus musculus
The gene expression of an orthotopic mouse model developed from human glioblastoma neurospheres was compared between temozolomide treated and untreated mice. Overall design: Sampling consisted of seve
NIAID Data Ecosystem20
snRNA-seq of human cutaneous neurofibromas before and after selumetinib treatment implicates role of altered Schwann cell states, inter-cellular signaling, and extracellular matrix in treatment response. snRNA-seq of human cutaneous neurofibromas before and after selumetinib treatment implicates role of altered Schwann cell states, inter-cellular signaling, and extracellular matrix in treatment response
Neurofibromatosis Type I is caused by loss of function variants in the NF1 gene. Most patients with NF1 develop skin lesions called cutaneous neurofibromas (cNFs). Currently the only approved therapeu
NIAID Data Ecosystem00



