Characterization of a spontaneous model of inflammation-derived liver cancer [ChIP-seq]. Mus musculus
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA319967
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Induction of a protective response to oxidative stress, both from a transcriptional and from a genetic viewpoint, confers a selective advantage for cells exposed to an inflammatory environment, paving the way to neoplastic transformation Overall design: Chromatin immuno-precipitations of acetylated lysine 27 of H3 followed by multiparallel sequencing, performed on tissues of Mdr2-WT (FVB.129P2) livers, Mdr2-KO (FVB.129P2-Abcb4tm1Bor/J) inflamed livers or hepatocarcinoma nodules from Mdr2-KO livers. All samples have been treated with clodronate liposomes to prevent sample contamination by liver macrophages
创建时间：
2016-04-28
