首页/数据集/FoxA1 and FoxA2 regulate growth and cellular identity in NKX2-1-positive lung adenocarcinoma. FoxA1 and FoxA2 regulate growth and cellular identity in NKX2-1-positive lung adenocarcinoma

FoxA1 and FoxA2 regulate growth and cellular identity in NKX2-1-positive lung adenocarcinoma. FoxA1 and FoxA2 regulate growth and cellular identity in NKX2-1-positive lung adenocarcinoma

NIAID Data Ecosystem2026-03-13 收录

肺腺癌分子机制

转录因子调控

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA778778 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

应用场景：

创建时间：

2021-11-08

黑色素细胞

转录因子调控

Mutations in the gene encoding transcription factor TFAP2A result in pigmentation anomalies in model organisms and premature hair graying in humans. However, the pleiotropic functions of TFAP2A and it

NIAID Data Ecosystem60

Trasncripional regulation by overexpression of constitutively active FOXO1 (CA-FOXO1) in human endothelial cells. Trasncripional regulation by overexpression of constitutively active FOXO1 (CA-FOXO1) in human endothelial cells

转录因子调控

内皮细胞基因表达

To identify genes regulated by FOXO1, we performed RNA-seq analysis of human unbilical vein endothelial cells (HUVECs) overexpressing CA-FOXO1 by adenovirus. CA-FOXO1 is constructed by mutating three

NIAID Data Ecosystem40

Loss of thymidine kinase 1 inhibits lung cancer growth and metastatic attributes by reducing GDF15 expression. Homo sapiens

肺腺癌分子机制

肿瘤靶点发现

Metabolic alterations that are critical for cancer cell growth and metastasis are one of the key hallmarks of cancer. Here, we show that thymidine kinase 1 (TK1) is significantly overexpressed in tumo

NIAID Data Ecosystem30

DataSheet_1_YTHDF2 Inhibits the Migration and Invasion of Lung Adenocarcinoma by Negatively Regulating the FAM83D-TGFβ1-SMAD2/3 Pathway.pdf

肺腺癌分子机制

癌症信号通路

ObjectiveYTH domain family 2 (YTHDF2) is an important N6-methyladenosine (m6A) reader, but its role in lung adenocarcinoma remains elusive. This study assessed its function in lung adenocarcinoma. Met

NIAID Data Ecosystem40

anti-Eomes ChIPseq of control OT-I cells and Eomes-overexpressing OT-I cells. anti-Eomes ChIPseq of control OT-I cells and Eomes-overexpressing OT-I cells

转录因子调控

T细胞耗竭

High amount of Eomes might drive T cell exhaustion. In order to understand how Eomes contributes to exhaustion of CD8+ T cell in the TME as a transcription factor, we conducted anti-Eomes ChIPseq anal

NIAID Data Ecosystem30

FoxA1 and FoxA2 regulate growth and cellular identity in NKX2-1-positive lung adenocarcinoma. FoxA1 and FoxA2 regulate growth and cellular identity in NKX2-1-positive lung adenocarcinoma

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