Low KLF11 Determines the CSC–like Identity in Sarcoma via restraining YAP transactivity [ChIP-seq]
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE133310
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资源简介：
We conducted ChIP-seq experiments to investigate the role of KLF11 in YAP/TEAD transcriptional activity. ChIP-Seq profiles for KLF11, YAP, TEAD1, SIN3A, and HDAC2 in MG63 cells. ChIP-Seq profiles for H3K27ac in MG63 cells transfected with KLF11 or control siRNA. ChIP-Seq profiles for KLF11 in MG63 cells transfected with YAP or control siRNA. BioProject accession is PRJNA549158
创建时间：
2021-02-23
