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Dissecting placental host-pathogen interactions: Rift Valley fever virus infection in early human trophoblast stem cells

ChEBI2025-12-12 更新2026-07-12 收录

胎盘病毒学

裂谷热病毒感染机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-16352 数据链接链接失效反馈

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资源简介：

Dissecting placental host-pathogen interactions: Rift Valley fever virus infection in early human trophoblast stem cells

应用场景：

创建时间：

2025-12-12

裂谷热病毒感染机制

抗病毒药物靶点

As an arthropod-borne human pathogen, Rift Valley fever virus (RVFV) cycles between an insect vector and mammalian hosts. Little is known about the cellular requirements for infection in either host.

NIAID Data Ecosystem60

DDX17 CLIP-seq in U2OS cells with and without Rift Valley Fever Virus infection.. Homo sapiens strain:U2OS

RNA-蛋白互作

裂谷热病毒感染机制

DDX17 CLIP-seq in Rift Valley Fever Virus infected and uninfected human U2OS cells.

NIAID Data Ecosystem40

DataSheet_1_Inefficient Placental Virus Replication and Absence of Neonatal Cell-Specific Immunity Upon Sars-CoV-2 Infection During Pregnancy.pdf

胎盘病毒学

围产期免疫

Pregnant women have been carefully observed during the COVID-19 pandemic, as the pregnancy-specific immune adaptation is known to increase the risk for infections. Recent evidence indicates that even

NIAID Data Ecosystem10

Respiratory syncytial virus exhibits differential tropism for distinct human placental cell types with Hofbauer cells acting as a permissive reservoir for infection

胎盘病毒学

母婴垂直传播

Background Respiratory syncytial virus (RSV) is capable of transient viremia and extrapulmonary dissemination. Recently, this virus has been identified in fetal cord blood, suggesting the possibility

NIAID Data Ecosystem50

Comparison of MBT/Pas and BALB/cByJ MEFs response after infection with Rift Valley Fever virus

裂谷热病毒感染机制

宿主遗传抗性研究

The Rift Valley Fever (RVF) is an arthropod-borne disease present in several countries of Africa and Middle East. It is caused by RVF virus which can infect both humans and animals. In humans, it lead

NIAID Data Ecosystem20

Dissecting placental host-pathogen interactions: Rift Valley fever virus infection in early human trophoblast stem cells

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