Related Article from Tumor Endothelial Cells Join the Resistance

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Related Article from Tumor Endothelial Cells Join the Resistance

创建时间：

2009-08-01

Single Cell RNA Sequencing of WT and Cdh5-cre Myct1 fl/fl Endothelium

肿瘤内皮细胞

单细胞转录组学

Edothelial cells were FACS sorted as CD31+CD45- population from PYMT-BO1 transplant tumor bearing WT and Cdh-cre Myct1 fl/fl mice on Day 14 post tumor transplantation Overall design: Single-cell RNA-s

NIAID Data Ecosystem60

Genome-wide maps of WT and over-expressing CenH3/CENP-A in Human HeLa S3 cells. Homo sapiens

表观遗传调控

肿瘤治疗抵抗

Centromeres are essential to ensure proper chromosome segregation in eukaryotes. Their definition relies on the presence of a centromere-specific H3 histone variant CenH3, known as CENP-A in mammals.

NIAID Data Ecosystem30

The induction of core pluripotency master regulators in cancers defines poor clinical outcomes and treatment resistance

肿瘤治疗抵抗

多能性调控因子

Clinical expression of core pluripotent stem cell master regulators OCT4, SOX2 and NANOG is associated with treatment resistance and lethal cancers. We have described a novel model to induce the expre

NIAID Data Ecosystem30

Transcriptional Inactivation of TP53 and BMP Pathway Components Mediates Therapy-induced Dedifferentiation and Metastasis in Prostate Cancer. Transcriptional Inactivation of TP53 and BMP Pathway Components Mediates Therapy-induced Dedifferentiation and Metastasis in Prostate Cancer

前列腺癌转移

肿瘤治疗抵抗

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem40

Expression profiling of tumor lymphatic endothelial cells upon acute Ang2 inhibition. Expression profiling of tumor lymphatic endothelial cells upon acute Ang2 inhibition

肿瘤内皮细胞

信号通路抑制

Lymphatic endothelial cells (LEC) were isolated from MT-ret tumor tissue 12h after treatment with an Ang2-blocking antibody or control-IgG Overall design: Total RNA-sequencing of FACS-sorted LEC. Five

NIAID Data Ecosystem30

Related Article from Tumor Endothelial Cells Join the Resistance

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