Crosstalk between the STAT3, NF-kB and Notch signaling pathways in glioblastoma stem cells

ChEBI2013-06-19 更新2026-07-21 收录

胶质母细胞瘤分子机制

信号通路交互研究

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Crosstalk between the STAT3, NF-kB and Notch signaling pathways in glioblastoma stem cells

创建时间：

2013-06-19

Enhancer profiling of glioblastoma uncovers core oncogenic dependency and therapeutic opportunity [ChIP-seq]

胶质母细胞瘤分子机制

增强子分析

Here, by mapping H3K27ac deposition, we analyze the active regulatory landscapes across primary GBM biopsies, normal brain tissues, and cell line counterparts. Analysis of differentially regulated enh

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Title RNA-seq of human glioblastoma cells in response to ITE treatment

胶质母细胞瘤分子机制

单细胞RNA测序药物研究

Aryl hydrocarbon receptor (AhR) is a ligand-activated and cancer releated transcription factor. 2-(1'H-indole-3'-carbonyl)-thiazole-4-carboxylic acid methyl ester (ITE) is an AhR ligand, which regulat

NIAID Data Ecosystem50

Cellular senescence in malignant cells promotes tumorigenesis in mouse and patient glioblastoma [RNA-seq]. Cellular senescence in malignant cells promotes tumorigenesis in mouse and patient glioblastoma [RNA-seq]

胶质母细胞瘤分子机制

衰老转录组学

We performed a scRNAseq analysis conducted at the endpoint of our GBM mouse model. We compared differentially expressed up-regulated genes in p16Ink4a positive vs p16Ink4a negative malignant cells. Th

NIAID Data Ecosystem30

Expression data from glioblastoma tissue and matched adjacent normal tissue. Homo sapiens

胶质母细胞瘤分子机制

癌细胞转录组

Glioblastoma (GBM) is the most common and lethal primary malignancy of the central nervous system in adult. In order to improve the diagnosis, prevention and treatment of GBM, the details of molecular

NIAID Data Ecosystem50

Additional file 12 of Decoding key cell sub-populations and molecular alterations in glioblastoma at recurrence by single-cell analysis

胶质母细胞瘤分子机制

细胞基因调控网络

Additional file 12. Table S7. The intersection of rGBM GS genes and MAFK targets from ChIP-Atlas.

Figshare2023-07-31 更新40