MiR-210 is induced by Oct-2, regulates B-cells and inhibits autoantibody production

ChEBI2013-06-21 更新2026-07-21 收录

免疫调节分子机制

细胞功能调控

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-48186 数据链接链接失效反馈

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MiR-210 is induced by Oct-2, regulates B-cells and inhibits autoantibody production

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2013-06-21

CD73 Immune Checkpoint Defines Inducible Regulatory NK cells in the Tumor Microenvironment

肿瘤免疫微环境

细胞功能调控

High levels of ecto-5'-nucleotidase (CD73) have been implicated in immune suppression and tumor progression. Elevated levels of CD73 has also been observed in patients who progress on anti–PD-1 immuno

NIAID Data Ecosystem90

Cytokine expression in lymph nodes of WT and Lcn-2 KO mice 7 days after NTS induction.

免疫调节分子机制

基因表达分析

WT (n = 13) and Lcn-2 KO mice (n = 12) were subjected to NTS and followed for 7 days. cDNA isolated from lymph nodes was analyzed for the expression of the respective genes. The respective gene/HPRT r

NIAID Data Ecosystem50

Human Tumour Immune Evasion via TGF-β Blocks NK Cell Activation but Not Survival Allowing Therapeutic Restoration of Anti-Tumour Activity

免疫逃逸机制

细胞功能调控

Immune evasion is now recognized as a key feature of cancer progression. In animal models, the activity of cytotoxic lymphocytes is suppressed in the tumour microenvironment by the immunosuppressive c

Figshare2016-01-18 更新40

PEST-domain-enriched tyrosine phosphatase and glucocorticoids as regulators of mast cell signalling

细胞信号通路

免疫调节分子机制

PEST-domain-enriched tyrosine phosphatase (PEP) is a cytoplasmic protein tyrosine phosphatase that regulates immune cell functions, including mast cell functions. Using bone marrow derived mast cells

NIAID Data Ecosystem90

Evaluating the role of individual NK cell ligands in HIV-specific recognition by NK cells

HIV免疫识别机制

细胞功能调控

In our previous experiments, we performed phenotypic analysis on NK cells that had been co-cultured with HIV-infected or mock-infected CD4+ T cells to identify the NK cell receptors that are predictiv

NIAID Data Ecosystem40

MiR-210 is induced by Oct-2, regulates B-cells and inhibits autoantibody production

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